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NCI-H716 cells as a model for endocrine differentiation in colorectal cancer
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
Uncoupling protein 2 regulates glucagon-like peptide-1 secretion in L-cells
CGA immunoreactivity in NCI-H716 cells in varous conditions. A:... | Download Scientific Diagram
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FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
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RT-PCR of transcription factor expression in NCI-H716 cells. RNA was... | Download Scientific Diagram
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Figure S1, FGFR2 copy gain and overexpression in H716 cells A - ppt download
Sennoside A Induces GLP-1 Secretion Through Activation of the ERK1/2 P | DMSO
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE
Peptide production and secretion in GLUTag, NCI-H716, and STC-1 cells: a comparison to native L-cells in: Journal of Molecular Endocrinology Volume 56 Issue 3 (2016)
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
The ginsenoside metabolite compound K stimulates glucagon-like peptide-1 secretion in NCI–H716 cells by regulating the RhoA/ROCKs/YAP signaling pathway and cytoskeleton formation - ScienceDirect
NCI-H716 [H716] - CCL-251 | ATCC
FGFR2 Is Amplified in the NCI-H716 Colorectal Cancer Cell Line and Is Required for Growth and Survival | PLOS ONE